Structure, Form, and Neurophysiology in Criminal Desistance

David Phillips • September 6, 2024
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Introduction

As an undergraduate student at the University of Pittsburgh, I became interested in experimental functional morphology, particularly the effects of soft tissue functionality on hard tissue structures. Recently, it occurred to me that principles of functionality are apparent in sociology, particularly in criminal desistance. Criminal desistance, the process through which individuals cease engaging in criminal behavior, has garnered significant interest in criminology, psychology, and neurophysiology. Understanding the structural and formational elements of desistance and their neurophysiological underpinnings can provide insights into the mechanisms that facilitate this complex process.


Criminal Desistance: An Overview

Criminal desistance is a multifaceted process influenced by various factors, including social structures, individual agency, and biological underpinnings. While traditional criminological theories have emphasized the role of social and environmental factors, recent advances in neurophysiology offer new perspectives on how brain structures and functions may influence an individual's propensity to desist from crime. This paper aims to integrate these perspectives, providing a comprehensive understanding of the structural, formational, and neurophysiological aspects of criminal desistance.

Desistance has two forms of structure: social and physiological.


Structure and Its Role in Criminal Desistance


Social Structure and Environmental Influences

Social structure refers to the organized patterns of relationships and institutions that shape an individual's behavior and opportunities. Key structural factors influencing criminal desistance include:

  1. Family and Social Bonds: Strong, supportive relationships with family members and prosocial peers are critical for desistance. Positive social bonds provide emotional support, guidance, and a sense of belonging, which can deter individuals from returning to criminal activities (Laub & Sampson, 2001).
  2. Employment and Education: Access to stable employment and educational opportunities can enhance self-efficacy and provide legitimate means for achieving personal goals. These opportunities help individuals build a positive identity and reduce reliance on criminal activities for economic survival (Uggen, 2000).
  3. Community and Environmental Context: Living in a supportive and resource-rich community can facilitate desistance by providing access to services, reducing exposure to criminogenic influences, and fostering a sense of community cohesion and safety (Kubrin & Weitzer, 2003).


Physiological Structure

We know that both genetic and biological factors can influence human behaviors.  Neurodevelopmental research indicates that social interactions with peers, authorities, and influencers are crucial in shaping neural pathways in the brain's prefrontal cortex that promote moral decision-making (Welborn et al., 2016).


Form: Individual Agency and Identity in Criminal Desistance


Form refers to the individual's capacity for agency, self-concept, and identity transformation. Critical aspects of form that influence criminal desistance include:

  1. Cognitive Transformation: Desistance often involves a cognitive shift in how individuals perceive themselves and their futures. This shift may include adopting a non-offender identity, reevaluating past behaviors, and setting new life goals (Giordano, Cernkovich, & Rudolph, 2002).
  2. Motivation and Intentionality: The decision to desist from crime is often driven by a combination of intrinsic and extrinsic motivations. Personal aspirations, the desire for a better life, and external pressures (such as legal consequences or family expectations) can all play a role in fostering desistance (Maruna, 2001).
  3. Self-Control and Decision-Making: Enhanced self-control and improved decision-making skills are crucial for maintaining desistance. These skills enable individuals to resist temptations, manage stress, and make choices aligned with their long-term goals (Moffitt, 1993).


Neurophysiology: Biological Underpinnings of Desistance


Advances in neurophysiology provide insights into how brain structures and functions may influence criminal behavior and desistance. Key neurophysiological factors include:

  1. Prefrontal Cortex Functioning: The prefrontal cortex (PFC) is involved in executive functions, such as impulse control, planning, and decision-making. Enhanced PFC functioning is associated with better self-control and resisting criminal impulses (Raine, 2013).
  2. Neuroplasticity and Rehabilitation: Neuroplasticity refers to the brain's ability to reorganize itself by forming new neural connections. Rehabilitation programs that promote cognitive-behavioral changes can harness neuroplasticity to facilitate desistance by strengthening neural pathways associated with prosocial behaviors (Davidson & McEwen, 2012).
  3. Neurochemical Influences: Neurotransmitters, such as serotonin and dopamine, play a role in regulating mood, motivation, and behavior. Imbalances in these neurochemicals can contribute to impulsivity and aggression, whereas therapeutic interventions that address these imbalances may support desistance (Buckholtz & Meyer-Lindenberg, 2012).


Integrative Framework for Understanding Criminal Desistance


To understand criminal desistance comprehensively, it is essential to integrate the structural, formational, and neurophysiological perspectives. An integrative framework highlights the dynamic interplay between social environments, individual agency, and biological factors:

  1. Interaction between Structure and Form: Social structures can shape individual agency by providing opportunities and constraints. Conversely, individuals can influence their environments through their actions and decisions. For example, securing stable employment (a structural factor) can enhance self-efficacy and identity transformation (formational factors), which in turn supports desistance (Sampson & Laub, 1993).
  2. Neurophysiological Mediators: Neurophysiological factors can mediate the relationship between social structures and individual agency. For instance, improved prefrontal cortex functioning may enhance decision-making skills, enabling individuals to take advantage of social opportunities and resist criminogenic influences (Piquero, 2008).
  3. Feedback Loops: Desistance is a dynamic process with feedback loops. Positive changes in one domain (e.g., securing employment) can lead to improvements in other domains (e.g., cognitive transformation), creating a reinforcing cycle that supports long-term desistance (Laub & Sampson, 2001).


Conclusion

Criminal desistance is a complex process influenced by social structures, individual forms, and neurophysiological factors. By integrating these perspectives, we can develop a more comprehensive understanding of the mechanisms that facilitate desistance. This integrative approach can inform the design of more effective interventions and policies to support individuals in their journey away from crime.

References

Buckholtz, J. W., & Meyer-Lindenberg, A. (2012). MAOA and the neurogenetic architecture of human aggression. Trends in Neurosciences, 35(1), 17-27.

Davidson, R. J., & McEwen, B. S. (2012). Social influences on neuroplasticity: stress and interventions to promote well-being. Nature Neuroscience, 15(5), 689-695.

Giordano, P. C., Cernkovich, S. A., & Rudolph, J. L. (2002). Gender, crime, and desistance: Toward a theory of cognitive transformation. American Journal of Sociology, 107(4), 990-1064.

Kubrin, C. E., & Weitzer, R. (2003). New directions in social disorganization theory. Journal of Research in Crime and Delinquency, 40(4), 374-402.

Laub, J. H., & Sampson, R. J. (2001). Understanding desistance from crime. Crime and Justice, 28, 1-69.

Maruna, S. (2001). Making good: How ex-convicts reform and rebuild their lives. American Psychological Association.

Moffitt, T. E. (1993). Adolescence-limited and life-course-persistent antisocial behavior: A developmental taxonomy. Psychological Review, 100(4), 674-701.

Piquero, A. R. (2008). Taking stock of developmental trajectories of criminal activity over the life course. In The long view of crime: A synthesis of longitudinal research (pp. 23-78). Springer, New York, NY.

Raine, A. (2013). The anatomy of violence: The biological roots of crime. Pantheon Books.

Sampson, R. J., & Laub, J. H. (1993). Crime in the making: Pathways and turning points through life. Harvard University Press.

Uggen, C. (2000). Work as a turning point in the life course of criminals: A duration model of age, employment, and recidivism. American Sociological Review, 65(4), 529-546.

Welborn, B. L., Lieberman, M. D., Goldenberg, D., Fuligni, A. J., Galván, A., & Telzer, E. H. (2016). Neural mechanisms of social influence in adolescence. Social Cognitive and Affective Neuroscience, 11(1), 100-109. Doi: 10.1093/scan/nsv095. Epub 2015 Jul 21. PMID: 26203050; PMCID: PMC4692320.

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